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As we age, NAD+ levels gradually decrease, resulting in decreased energy levels and physical function. Lower NAD+ levels can cause skin aging, decreased energy, less energy than before, decreased muscle levels, and can also cause a variety of diseases such as cardiovascular disease and neurodegenerative diseases.
NMN is the precursor of NAD+, and its function is reflected by converting it into NAD+ in the human body, thereby increasing the NAD+ level in the human body.
Mitochondria are the main "energy powerhouses" in cells, and most metabolic activities in cells require the use of energy stored in ATP. Mitochondria can store energy from food in ATP for subsequent use by metabolic activities. Supplementing NMN can increase the activity of NAD+-dependent Sirts enzymes, remove excessive acetylation modifications of mitochondrial proteins, regulate the activity of downstream metabolic enzymes, greatly improve the ability of mitochondria to provide energy, "nourish" a variety of tissues and organs, and resist aging.
Stem cells are a type of cells with self-renewal ability and differentiation potential. Stem cells continue to proliferate, with some remaining stem cells and maintaining self-renewal; the other part differentiates to replenish various tissues and organs. NMN can provide supplementary energy to a variety of stem cells, activate proliferation-related pathways, and promote the proliferation of a variety of stem cells.
Oxidative stress refers to the imbalance between oxidation and antioxidant effects in the body, which tends to oxidize, leading to inflammatory infiltration of neutrophils, increased secretion of proteases, and the production of a large number of oxidation intermediates. Oxidative stress is a negative effect caused by free radicals in the body. Almost all cells in the human body are easily damaged by oxidative stress. It is linked to many aging-related health problems, such as Alzheimer's disease and impaired blood sugar and heart function. NMN can improve cognitive function, reduce the level of mitochondrial oxidative stress in cardiomyocytes, and weaken the oxidative stress state in nerve cells.
*Special note - This article is for informational purposes only and cannot replace a doctor's treatment diagnosis and advice. It should not be regarded as a recommendation or proof of efficacy of the medical products involved. If it involves disease diagnosis, treatment, and rehabilitation, please be sure to go to a professional medical institution to seek professional advice.
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